Modul 1General Pathomorphology Text tests



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  • Flebits

  • Organization

  • Local hyperemia hepatic venous thrombosis occurs when:

    1. * Hepatic veins

    2. Hepatic artery

    3. Portal vein

    4. Kava-cava anastomoses

    5. Porto-cava anastomoses

  • Name gastric bleeding:

    1. Haemorrhagіa per rexіn

    2. Haemoptoe

    3. haemorrhagіa per dіabrosіn

    4. Haematemesіs

    5. * Maelena

  • Name of the macroscopic clot

    1. Erythrocytes, fibrin

    2. Leucocytes, platelets

    3. * The head, body, tail

    4. The inclusion of calcium

    5. Pigments

  • Name the author triad thrombotic

    1. * Virhov

    2. Apricot

    3. Rusakov

    4. Minakov

    5. Davidovsky

  • Name the most common type of embolism in hospital

    1. Bacterial

    2. Aerial

    3. Gas

    4. The fatty

    5. * Thromboembolism

  • Please indicate what elements make up a mixed thrombus

    1. * Erythrocytes, leukocytes, fibrin

    2. Leucocytes, fibrin, plasma proteins

    3. Fibrin platelets

    4. Plasma proteins, erythrocytes

    5. Platelets, erythrocytes

  • Please indicate what elements make up a white thrombus

    1. Plasma proteins

    2. * Erythrocytes, fibrin, platelets

    3. Fibrin, leukocytes

    4. Leucocytes, platelets,

    5. Erythrocytes

  • Red thrombus frequently occur in:

    1. * Veins

    2. Aorta

    3. Pulmonary artery

    4. Arterioles

    5. Kappilary

  • ?Reduced content of tissue fluid is called:

    1. * Dehydration

    2. Destruction

    3. Degeneration

    4. Detoxication.

    5. Deprivation

  • Relating to the hemodynamic changes in the shock:

    1. Arterial hyperemia

    2. * Sladzh-syndrome

    3. Vakatnaya hyperemia

    4. Inflammatory hyperemia

    5. Venous bleeding

  • Retrograde embolism characteristic:

    1. * Embolism by outside bodies

    2. Embolism microbes

    3. Fat embolism

    4. Air embolism

    5. Gas embolism

  • Specify the macroscopic part of clot

    1. Erythrocytes, fibrin

    2. Leucocytes, platelets

    3. * The head, body, tail

    4. The inclusion of calcium

    5. Pigments

  • Specify the most common type of embolism in hospital

    1. Bacterial

    2. Aerial

    3. Gas

    4. The fatty

    5. * Thromboembolism

  • Specify the type of clot, depending on its composition:

    1. Organized by

    2. * White, red

    3. On the corrugated surface

    4. Yellow

    5. Parietal

  • Specify the type of local arterial pathological hyperemia:

    1. * Angyonevrotic

    2. Dystrophic

    3. Emotional

    4. Discirculatoryty

    5. Arterial hypertension

  • Specify the type of local arterial pathological hyperemia:

    1. Working

    2. Dystrophic

    3. * Collateral

    4. --

    5. Arterial hypertension

  • Stagnant edema develops when:

    1. * Throm

    2. Stagnation of bile

    3. Hypodynamy

    4. Stagnation of urine

    5. Tromboarteriolite

  • Stasis - it stops the current of blood in:

    1. Aorta

    2. Artery

    3. Hepatic veins

    4. Veins

    5. * Kappilary

  • That becomes the skin in chronic venous hyperemia:

    1. * Cyanosis

    2. Red

    3. Ikteric

    4. Gray

    5. Pale

  • The accumulation of fluid in the abdominal cavity is called:

    1. Agenezia

    2. Ascytis

    3. Gydroperikard

    4. Sarcoma

    5. Hypertension

  • The accumulation of fluid in the emptiness of the heart called the shirts:

    1. Gidrotoraks

    2. Ageneziya

    3. Gidrotoraks

    4. Sarcoma

    5. * Gidroperikard

  • The accumulation of fluid in the main brain called:

    1. * Hydrocephalus

    2. Agenezia

    3. Ascytis

    4. Sarcoma

    5. Hypertension.

  • The accumulation of fluid in the pleural emptiness is called:

    1. * Gidrotoraks

    2. Ageneziya

    3. Gidroperikard

    4. Sarcoma

    5. Hypertension

  • The accumulation of fluid in the subcutaneous tissue is called:

    1. * Anasarca

    2. Ageneziya

    3. Gipervolemiya

    4. Sarcoma

    5. Hypertension

  • The cause of sclerotic changes in chronic venous hyperemia is:

    1. * Hypoxia

    2. Glykogenosis

    3. Hyalinosis

    4. Gydrotorax

    5. Hypertrophy

  • The main morphological manifestation of

    1. Postponing amyloid

    2. Postponing calcium salts

    3. * Growth of connective tissue

    4. Mucus

    5. Vascularization

  • The more complicated the shin deep vein thrombosis?

    1. Myocardial infarction

    2. Lung gangrene

    3. Gangrene bottom

    4. Gangrene intestine

    5. * Thromboembolism branch pulmonary artery

  • The most dangerous for the body is swelling:

    1. Bone marrow

    2. Subcutaneous tissue

    3. Lower limbs

    4. Skeletal

    5. * Main Brain

  • The most dangerous for the body is swelling:

    1. * Light

    2. Subcutaneous tissue

    3. Lower limbs

    4. Skeletal

    5. Bone marrow

  • The most frequently ICE syndrome develops when:

    1. Chronic pneumonia

    2. Cardiosclerosis

    3. Chronic gastritis

    4. * Premature detachment of placenta and amniotic fluid embolism

    5. Chronic gastritis

  • There are the following local pathological arterial hyperemia:

    1. * Angyonevrotic

    2. Dystrophic

    3. Emotional

    4. Discirculatoryty

    5. Arterial hypertension

  • There are the following local pathological arterial hyperemia:

    1. Working

    2. Dystrophic

    3. * Collateral

    4. --

    5. Arterial hypertension

  • Thrombosis - is:

    1. Circulation in the blood formed elements

    2. * Lifetime blood clotting in the heart cavities and in the lumen of vessels

    3. Blood clotting in the serous cavities

    4. Post-mortem blood clotting

    5. Stop bleeding

  • Thrombosis in the right atrium via tromboembolicheskie can lead to complications:

    1. Venous stagnation in the lungs

    2. myocardial infarction

    3. Dilatation of the heart ventricles

    4. brain infarction

    5. * Pulmokoronary shock

  • Thrombotic triad called the author:

    1. * Virhov

    2. Apricot

    3. Rusakov

    4. Minakov

    5. Davidovsky

  • Total venous plethora of pathology occurs when:

    1. Main Brain

    2. Heart

    3. Liver

    4. Aorta

    5. Hollow vein

  • Types clot, depending on its composition

    1. Organized by

    2. * White, red

    3. On the corrugated surface

    4. Yellow

    5. Parietal

  • Types clot, depending on its composition

    1. Organized by

    2. White with gemmorhagical rim

    3. Obturating

    4. Yellow

    5. * Mixed

  • Unfavorable results investigation thrombosis

    1. Vascularization

    2. Stasis

    3. * Purulent fusion

    4. Aseptic autolysis

    5. embolism

  • Varicose veins of esophagus occurs when the blood on the current difficult :

    1. Veins light

    2. Hepatic arteries

    3. * Portal veins

    4. Esophageal veins

    5. Esophageal arteries

  • Varicose veins of lower extremities may be difficult tromboembol:

    1. Portal vein

    2. Pulmonary veins

    3. Mesenteric arteries

    4. * Pulmonary artery

    5. Bifurcation of the aorta

  • What are swelling in the tissues:

    1. * Stagnation

    2. Hyaline

    3. Atrophic

    4. Glikogennye

    5. Hypertrophic

  • What are the mechanism of development of renal edema with glomerulonephritis:

    1. * Delayed sodium

    2. Hypoxia

    3. The delayed potassium

    4. Hypodynamia

    5. Hypertension

  • What belongs to the morphological manifestation of shock:

    1. * Liquid blood

    2. Reduced blood

    3. Muscat liver

    4. The accumulation of blood in heart

    5. Glikogen

  • What can complicate deep vein thrombosis ?

    1. Myocardial infarction

    2. Lung gangrene

    3. Gangrene bottom

    4. Gangrene intestine

    5. * Thromboembolism branch pulmonary artery

  • What cause the appearance of blood clots in the aortic aneurysm

    1. * Violation of the integrity of intima

    2. Thromboembolism

    3. Venous stagnation

    4. Expansion vessel

    5. Necrosis

  • What dystrophy develops in the liver in chronic venous hyperemia:

    1. Mucosal

    2. Mucosal

    3. * The fatty

    4. Mineral

    5. Carbohydrate

  • What edema is the most dangerous for the organism:

    1. Bone marrow

    2. Subcutaneous tissue

    3. Lower limbs

    4. Skeletal

    5. * Main Brain

  • What edema is the most dangerous for the organism:

    1. * Light

    2. Subcutaneous tissue

    3. Lower limbs

    4. Skeletal

    5. Bone marrow

  • What elements make up a mixed thrombus

    1. * Erythrocytes, leukocytes, fibrin

    2. Leucocytes, fibrin, plasma proteins

    3. Fibrin platelets

    4. Plasma proteins, erythrocytes

    5. Platelets, erythrocytes

  • What elements make up a white thrombus

    1. Plasma proteins

    2. * Erythrocytes, fibrin, platelets

    3. Fibrin, leukocytes

    4. Leucocytes, platelets,

    5. Erythrocytes

  • What is a heart attack:

    1. * Vascular necrosis

    2. Toxic necrosis

    3. Allergic necrosis

    4. Traumatic necrosis

    5. Mixed necrosis

  • What is a retrograde embolism:

    1. Movement embol through open oval ring

    2. * Movement embol against the current of blood

    3. Movement embol current blood

    4. Movement embol through артеріо-venous anastomoses

    5. Movement embol on lymphatic

  • What is considered a retrograde embolism:

    1. * Embolism by outside bodies

    2. Embolism microbes

    3. The fatty embolism

    4. Air embolism

    5. Gas embolism

  • What is manifested acute venous hyperemia:

    1. * Plazmorhagy

    2. Hyalinosis

    3. Sclerosis

    4. Leukoplakia

    5. Glykogenosis

  • What is paradoxical embolism

    1. Movement embol against movement of blood movement embol current blood

    2. * Movement embol a small circle in a large, light bypassing

    3. Stop motion embol

    4. Slow motion embol

    5. Movement embol current blood

  • What is stasis - is stopping power in the blood:

    1. Aorta

    2. Artery

    3. Hepatic veins

    4. Veins

    5. * Kappilary

  • What is the benefit of a pleasant consequence of a heart attack:

    1. Atrophy

    2. Embolism

    3. Miomalacia

    4. * Scarring

    5. Hyperplasia

  • What is the cause of sclerotic changes in chronic venous hyperemia:

    1. * Hypoxia

    2. Glykogenosis

    3. Hyalinosis

    4. Gydrotorax

    5. Hypertrophy

  • What is the mechanism of development dystrophic edema:

    1. * Reduced pressure

    2. Hypoxia

    3. Increasing pressure

    4. Hypodynamia

    5. Hypertension

  • What is the mechanism of development of renal edema with glomerulonephritis:

    1. * Delayed sodium

    2. Hypoxia

    3. The delayed potassium

    4. Hypodynamia

    5. Hypertension

  • What is the name of bleeding due to rupture the wall of the vessel or heart:

    1. * Haemorrhagіa per rexіn

    2. Haemoptoe

    3. haemorrhagіa per dіabrosіn

    4. Haematemesіs

    5. Maelena

  • What is the name of bleeding from the lungs:

    1. * Haemoptoe

    2. Epіstaxіs

    3. Haemothorax

    4. Haematemesіs

    5. Maelena

  • What is the name of bleeding in the stomach:

    1. Haemorrhagіa per rexіn

    2. Haemoptoe

    3. haemorrhagіa per dіabrosіn

    4. Haematemesіs

    5. * Maelena

  • What is the name of reducing the content of tissue fluid:

    1. Detoxication.

    2. Destruction

    3. Degeneration

    4. * Dehydration

    5. Deprivation

  • What is the name of the accumulation of fluid in the subcutaneous tissue:

    1. Hypertension

    2. Ageneziya

    3. Gipervolemiya

    4. Sarcoma

    5. * Anasarca

  • What is the name of the accumulation of fluid in the emptiness of heart shirt:

    1. Gidrotoraks

    2. Ageneziya

    3. Gidrotoraks

    4. Sarcoma

    5. * Gidroperikard

  • What is the name of the accumulation of fluid in the pleural cavity:

    1. Sarcoma

    2. Ageneziya

    3. Gydroperikard

    4. * Gydrotoraks

    5. Hypertension

  • What is the name of the accumulation of fluid in the abdominal cavity:

    1. Agenezia

    2. Ascytis

    3. Gydropericard

    4. Sarcoma

    5. Hypertension

  • What is the name of the accumulation of fluid in the main brain:

    1. * Hydrocephalus

    2. Agenezia

    3. Ascytis

    4. Sarcoma

    5. Hypertension.

  • What kind of shock to distinguish etiology and Pathogenesis:

    1. * Traumatic

    2. Atrophic

    3. Gipotrophic

    4. Hypertrophic

    5. Hypertensive

  • What mechanism in the development of renal edema in nephrotic syndrome:

    1. * Gipoproteinemiya

    2. Hypoxia

    3. Hypotension

    4. Hypodynamia

    5. Hypertension

  • What mechanism in the development of renal edema in nephrotic syndrome:

    1. * Gipoproteinemiya

    2. Hypoxia

    3. Hypotension

    4. Hypodynamia

    5. Hypertension

  • What mechanism of development of dystrophic edema:

    1. * Reduced pressure

    2. Hypoxia

    3. Increasing pressure

    4. Hypodynamia

    5. Hypertension

  • What occurs in the body, with thrombosis of the artery

    1. Inflammation

    2. Edema

    3. Hyperemia

    4. Anemia

    5. * Infarction

  • What occurs in the tissues under anemia:

    1. Hypertrophy

    2. * Sclerosis

    3. Gaemomelanosis

    4. Hyperplasia

    5. Cyanosis

  • What refers to the beneficial effects of edema:

    1. Ossification fluid

    2. Organization of the liquid

    3. * Dispersal fluid

    4. Suppuration fluid

    5. Petrificats fluid

  • When hyperemia:

    1. High blood pressure

    2. Increasing the number of eritrocits

    3. * Improving barometric pressure

    4. Decreased blood pressure

    5. Increasing venous pressure

  • When a disease is found bacterial embolism

    1. Atherosclerosis

    2. Malignant tumors

    3. * Sepsis

    4. Bends

    5. Fractures

  • When anemia occurs in the tissues:

    1. Hypertrophy

    2. Sclerosis

    3. Gaemomelanosis

    4. Hyperplasia

    5. Cyanosis

  • When angionevrotic hyperemia:

    1. * Violation inervatsii receptacle

    2. Increasing the number of erytrotsits

    3. High blood pressure

    4. Decreased blood pressure

    5. Increasing venous pressure

  • When developing cardiac edema:

    1. * Koronarosklerosis

    2. Compensation heart

    3. Cardiac decompensation

    4. Increasing cardiac activity

    5. Cardiac hypertrophy

  • When developing stagnant edema:

    1. * Thromb

    2. Stagnation of bile

    3. Hypodynamy

    4. Stagnation of urine

    5. Tromboarteriolite

  • When ICE syndrome thrombus is more common in:

    1. Veins

    2. Emptiness of heart

    3. * Microvessels

    4. Aorta

    5. Great arteries

  • When in the ICE syndrome is often:

    1. * Necrosis

    2. Sclerosis

    3. Hyperplasia

    4. Atrophy

    5. Hypertrophy

  • When inflammatory hyperemia:

    1. * Increase the number of histamine in tissues

    2. Increasing the number of eritrotcits

    3. High blood pressure

    4. Decreased blood pressure

    5. Increasing venous pressure

  • When the engine develops a syndrome:

    1. * Premature detachment of placenta and amniotic fluid embolism

    2. Cardiosclerosis

    3. Chronic gastritis

    4. Chronic pneumonia

    5. Chronic gastritis

  • When the total venous hyperemia - in pathology:

    1. Main Brain

    2. Heart

    3. Liver

    4. Aorta

    5. Hollow vein

  • When there is a general acute venous hyperemia:

    1. * with myocardial infarction

    2. with cardiosclerosis

    3. with pneumosclerosis

    4. deficiencies in the heart

    5. with aortic atherosclerosis

  • When there is chronic venous hyperemia light:

    1. Pneumonia

    2. Mitral heart disease

    3. Emphysema

    4. Atherosclerosis

    5. Chronic bronchitis

  • When thromboembolism of the artery occurs pulmokoronarny shock:

    1. * Pulmonary artery

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