Modul 1General Pathomorphology Text tests



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calculous cholecystitis

  • abscess cholecystitis

  • purulent hepatitis hranulematoznoho

  • toxic liver dystrophy

  • * nehniynoho destructive (necrotizing) and cholangitis holanhiolitu

  • Primary hepatitis occurs following:

    1. bacteria

    2. mushrooms

    3. simpler

    4. Streptococci

    5. * Gepatotropnye viruses

  • Primary hepatitis occurs following:

    1. bacteria

    2. mushrooms

    3. simpler

    4. staphylococci

    5. * Alcohol

  • Primary hepatitis occurs following:

    1. bacteria

    2. mushrooms

    3. simpler

    4. Streptococci

    5. * drugs

  • Primary hepatitis occurs following:

    1. bacteria

    2. mushrooms

    3. simpler

    4. staphylococci

    5. * stagnation of bile

  • Primary incurred erosions heal over -

    1. 2-4 hours

    2. 4-6 hours

    3. 8-10 hours

    4. 12-18 hours

    5. * 24-48 hours

  • processes, EXCEPT:

    1. Atherosclerosis

    2. Xanthomas

    3. * Apoptosis

    4. Inflammation and necrosis

    5. Cholesterolosis.

  • Related to clinical changes with autoimmune gastritis is -

    1. Multiple erosions

    2. single erosion

    3. peptic ulcer

    4. mucosal atrophy

    5. * pernitsiozna anemia

  • Secondary biliary cirrhosis caused by:

    1. hepatitis

    2. alcoholic hepatitis

    3. active hepatitis

    4. excess copper in the blood

    5. * cholestasis due to extrahepatic biliary tract obstruction (stone, tumor)

  • Secondary biliary cirrhosis caused by:

    1. hepatitis

    2. alcoholic hepatitis

    3. active hepatitis

    4. chronic lead intoxication

    5. * biliary tract infection (holanhiolitychnyy cirrhosis)

  • Select from the above most accurate determination of erosion -

    1. swelling of the submucosa

    2. emergence phenomena intestinal metaplasia

    3. pronounced vasorelaxation mucosa

  • Select one of the listed diseases is an endogenous toxic gastritis -

    1. chronic bronchitis

    2. typhus

    3. esophageal cancer

    4. Viral hepatitis C

    5. * glomerulonephritis with the presence of uremia

  • Shnitslerovski metastasis of gastric cancer - a metastasis:

    1. mesenteric lymph nodes;

    2. parasternalni lymph nodes

    3. mediastinalni lymph nodes;

    4. left supraclavicular lymph nodes

    5. * adrectal fiber

  • Specific features of eosinophilic gastritis is -

    1. appearance kelyhopodibnyh cells

    2. mucosal atrophy and numerous eosinophil in inflammatory infiltrates

    3. mucosal erosion and numerous eosinophil in inflammatory infiltrates

    4. number of eosinophil in inflammatory infiltrates and edema formation pidslyzovoho

    5. * number of eosinophil in inflammatory infiltrates and mucus

  • Specific hepatic lipoproteins in viral hepatitis is formed by:

    1. reduction of virus in hepatocytes

    2. Restitution in holanhiotsytah virus and acts as antigen

    3. retraktsiyi virus in hepatocytes and acts as autoantibody

    4. virus infiltration nuclei of hepatocytes

    5. * viral replication in hepatocytes and acts as a self-antigen

  • The appearance of the stomach epitelioyidnoklitynnyh hranulom characteristic -

    1. eosinophilic gastritis

    2. gastric ulcer

    3. Zollinhera-Ellison syndrome

    4. reflux gastritis

    5. * sarcoidosis

  • The basis of cirrhosis is:

    1. dystrophy portal tracts

    2. necrosis of bile duct epithelium

    3. necrosis and regeneration of pathological bile duct epithelium

    4. hepatic vascular hyalinosis

    5. * dystrophy and necrosis of hepatocytes

  • The death of hepatocytes in cirrhosis leads to:

    1. Pathologic restitution portal tracts

    2. incomplete substitution of hepatocytes

    3. hyperplasia and hypertrophy of hepatic bile ducts

    4. liver infarctions

    5. * enhanced regeneration of stored parenchyma

  • The following features characterize irreversible cell injury, EXCEPT:

    1. Nucleous pyknosis

    2. Karyolysis

    3. Karyorrhaxis

    4. * Endoplasmic reticulum swelling

    5. Mitochondrial swelling.

  • The following features characterize reversible cell injury, EXCEPT:

    1. Blebs

    2. Endoplasmic reticulum swelling

    3. Dispersion of ribosomes

    4. * Myelin figures

    5. Autophagy

  • The group of inflammatory complications of peptic ulcer include:

    1. stenosis

    2. tetaniyu

    3. hlorhidropeniyu

    4. anemia

    5. * gastritis

  • The group of inflammatory complications of peptic ulcer include:

    1. stenosis

    2. tetaniyu

    3. hlorhidropeniyu

    4. anemia

    5. * Duodenitis

  • The group of inflammatory complications of peptic ulcer include:

    1. stenosis

    2. tetaniyu

    3. hlorhidropeniyu

    4. necrosis

    5. * peryduodenit

  • The group of ulcer complications, ulcer scar 12 - ring ulcer include:

    1. bleeding

    2. perforation

    3. Penetration

    4. metaplastychni processes

    5. * duodenal narrowing gap

  • The group-ulcer scar of gastric ulcer complications include:

    1. bleeding

    2. perforation

    3. Penetration

    4. narrowing of the esophagus

    5. * narrowing of the upper and the source of stomach

  • The group-ulcer scar of gastric ulcer complications include:

    1. bleeding

    2. perforation

    3. Penetration

    4. mucosal atrophy

    5. * deformation of the stomach

  • The liver in primary biliary cirrhosis increased and its surface:

    1. velykohorbysta

    2. velykozernysta

    3. posmuhovana

    4. bands of scar-

    5. * smooth or fine grained

  • The liver in primary biliary cirrhosis:

    1. not increased red

    2. increased brown-red

    3. increased yellow

    4. gray-red, reduced

    5. * increased in terms of gray-green

  • The liver in secondary biliary cirrhosis:

    1. not increased

    2. dough consistency

    3. increased soft

    4. gray-red, reduced

    5. * increased spacing

  • The liver in secondary biliary cirrhosis:

    1. brown due to hemosiderin

    2. red due to diapedesis erytrotstiv

    3. yellow color due to fat accumulation in hepatocytes

    4. gray with a red krapom

    5. * green due to bile soaking

  • The most common cause of fatty change in the heart is:

    1. Inflammation

    2. Neoplasia

    3. * Hypoxia

    4. Amyloidosis

    5. Autoimmune diseases.

  • The nephrotic syndrome is characterized by all of the following manifestations, EXCEPT:

    1. Massive proteinuria

    2. Hypoalbuminemia

    3. Generalized edema

    4. Hyperlipidemia and lipiduria

    5. * Hyperalbuminemia.

  • The stain used to identify fat is:

    1. Hematoxylin and eosin stain

    2. * Sudan III stain

    3. Congo red stain

    4. PAS reaction

    5. Metachromatic stain.

  • The stain used to identify glycogen is:

    1. Hematoxylin and eosin stain

    2. Sudan III stain

    3. Congo red stain

    4. * PAS reaction

    5. Metachromatic stain.

  • Acute total venous hyperemia occurs when:

    1. * Myocardial infarction

    2. Cardiosclerosis

    3. Pneumosclerosis

    4. Shortcomings of the heart

    5. Atherosclerosis of aorta

  • Acute total venous hyperemia occurs when:

    1. Shortcomings of the heart

    2. Cardiosclerosis

    3. Pneumosclerosis

    4. * Acute decompensation of heart

    5. Atherosclerosis of aorta

  • Acute total venous hyperemia occurs when:

    1. * Acute decompensation of heart

    2. Cardiosclerosis

    3. Pneumosclerosis

    4. Shortcomings of the heart

    5. Atherosclerosis of aorta

  • Acute venous hyperemia evident:

    1. * Plazmorhagy

    2. Hyalinosis

    3. Sclerosis

    4. Leukoplakia

    5. Glikogenosis

  • Air embolism occurs when the damage:

    1. Wen heads

    2. Wen esophagus

    3. * Wen neck

    4. Wan Light

    5. Wen abdominal

  • Angyonevrotic hyperemia occurs when:

    1. * Violation of innervation of the vessel

    2. Increasing the number of eritrotsitov

    3. High blood pressure

    4. Decreased blood pressure

    5. Increasing venous pressure

  • As a result of arterial thrombosis

    1. Inflammation

    2. Edema

    3. Hyperemia

    4. Anemia

    5. * Infarction

  • As often in the ICE syndrome with:

    1. * Necrosis

    2. Sclerosis

    3. Hyperplasia

    4. Atrophy

    5. Hypertrophy

  • Bleeding due to rupture the wall of the vessel or the heart is called:

    1. * Haemorrhagіa per rexіn

    2. Haemoptoe

    3. haemorrhagіa per dіabrosіn

    4. Haematemesіs

    5. Maelena

  • Bleeding from the lungs is called:

    1. * Haemoptoe

    2. Epіstaxіs

    3. Haemothorax

    4. Haematemesіs

    5. Maelena

  • By etiology, Pathogenesis and distinguish the following type of shock:

    1. * Traumatic

    2. Atrophic

    3. Gipotrophic

    4. Hypertrophic

    5. Hypertensive

  • By the favorable effects of edema include:

    1. Ossification fluid

    2. Organization of the liquid

    3. * Dispersal fluid

    4. Suppuration fluid

    5. Petrificats fluid

  • By the hemodynamic changes in the shock are:

    1. Arterial hyperemia

    2. * Sludge-syndrome

    3. Vakat hyperemia

    4. Inflammatory hyperemia

    5. Venous bleeding

  • By the morphological manifestations of the shock to:

    1. * Liquid blood

    2. Reduced blood

    3. Muscat liver

    4. The accumulation of blood in heart

    5. Glikogen

  • Cardiac edema develops when:

    1. * Cardiac decompensation

    2. Compensation heart

    3. Koronarosklerosis

    4. Increasing cardiac activity

    5. Cardiac hypertrophy

  • Chronic venous hyperemia lungs can occur if:

    1. Pneumonia

    2. Mitral heart disease

    3. Emphysema

    4. Atherosclerosis

    5. Chronic bronchitis

  • Death from shock pulmokoronary thromboembolism occurs when:

    1. * Pulmonary artery

    2. Aorta

    3. Aorta and coronary arteries

    4. Pulmonary veins

    5. Coronary artery

  • Definition of a paradoxal embolism

    1. Movement embol against movement of blood movement embola current blood

    2. * Movement embola a small circle in a large, light bypassing

    3. Stop motion embol

    4. Slow motion embol

    5. Movement embol current blood

  • Definition of retrograde embolism

    1. Movement embol through open oval ring

    2. * Movement embol against the current of blood

    3. Movement embol current blood

    4. Movement embol through artherio-venous anastomoses

    5. Movement embol on lymphatic

  • Definition ortograd embolism

    1. * Movement embol current blood

    2. Movement embol against the current of blood

    3. Movement embol a small circle in a large bypassing

    4. Movement embol on veins

    5. Embola Movement for arteries

  • Depending on the cause of edema in tissues are distinguished:

    1. * Stagnation

    2. Hyaline

    3. Atrophic

    4. Glikogennye

    5. Hypertrophic

  • Depending on the cause of edema in tissues are distinguished:

    1. * Kidney

    2. Hyaline

    3. Atrophic

    4. Glikogennye

    5. Hypertrophic

  • Describe mechanisms of development embolus

    1. The fatty dystrophy

    2. * Closing space vessels

    3. Pinocytosis

    4. Karyorhexis

    5. Protein dystrophy

  • Determination of thrombosis:

    1. Circulation in the blood formed elements

    2. * Lifetime blood clotting in the heart cavities and in the lumen of vessels

    3. Blood clotting in the serous cavities

    4. Post-mortem blood clotting

    5. Stop bleeding

  • Difficult blood flow in the veins which leads to varicose veins of esophagus:

    1. Veins light

    2. Hepatic arteries

    3. Portal veins

    4. Esophageal veins

    5. Esophageal arteries

  • Dilatation clot - thrombus it to:

    1. Left atrium

    2. * Aneurysm

    3. The right atrium

    4. The right ventricle

    5. Left ventricle

  • Enter the basic morphological manifestations of

    1. Postponing amyloid

    2. Postponing calcium salts

    3. * Growth of connective tissue

    4. Mucus

    5. Vascularization

  • Favorable consequence of a heart attack can be:

    1. Atrophy

    2. Embolism

    3. Miomalatia

    4. * Scarring

    5. Hyperplasia

  • Gialine thrombus formed in:

    1. * Kappilary

    2. The right atrium

    3. Left atrium

    4. Aorta

    5. Pulmonary artery

  • Give frequent hyaline localization of blood clots

    1. In the aorta

    2. In the coronary arteries

    3. In the hollows aortic aneurysm

    4. In the hepatic artery

    5. * In vessels microcirculatory channel

  • Give unfavorable results investigation thrombosis

    1. Vascularization

    2. Stasis

    3. * Purulent fusion

    4. Aseptic autolyze

    5. Fabric embolism

  • Heart attack - it is a fire:

    1. * Vascular necrosis

    2. Toxic necrosis

    3. Allergic necrosis

    4. Traumatic necrosis

    5. Mixed necrosis

  • How dangerous obstruktive thrombus?

    1. Povnokriv'ya

    2. * Of a heart attack

    3. Development of nephrotic syndrome

    4. Development of hemorrhagic syndrome

    5. Development of MS

  • Hyperemia occurs when:

    1. High blood pressure

    2. Increasing the number of eritrotsits

    3. * Improving barometric pressure

    4. Decreased blood pressure

    5. Increasing venous pressure

  • If the damage of the vein occurs air embolism:

    1. Wen heads

    2. Wen esophagus

    3. * Wen neck

    4. Wan Light

    5. Wen abdominal

  • In chronic venous hyperemia dystrophy develops in the liver:

    1. Mucosal

    2. Mucosal

    3. * The fatty

    4. Mineral

    5. Carbohydrate

  • In chronic venous hyperemia light becomes:

    1. * Brown

    2. Cyanotic

    3. Red

    4. Gray

    5. Pale

  • In chronic venous hyperemia liver becomes:

    1. Red

    2. Muscat

    3. Cyanosis

    4. Gray

    5. Pale

  • In chronic venous hyperemia observed:

    1. Tissue Atrophy

    2. Hyperplasia of tissues

    3. Hypertrophy

    4. Tissue necrosis

    5. * Tissue Hypoxia

  • In chronic venous hyperemia observed:

    1. Hyperplasia of tissues

    2. Sclerosis tissue

    3. Hypertrophy

    4. Tissue necrosis

    5. Tissue Hypoxia

  • In chronic venous hyperemia skin becomes:

    1. * Cyanosis

    2. Red

    3. Ikteric

    4. Gray

    5. Pale

  • In contrast to acute venous hyperemia, in chronic observed:

    1. Tissue Atrophy

    2. Hyperplasia of tissues

    3. Hypertrophy

    4. Tissue necrosis

    5. * Tissue Hypoxia

  • In contrast to acute venous hyperemia, in chronic observed:

    1. Hyperplasia of tissues

    2. Sclerosis tissue

    3. Hypertrophy

    4. Tissue necrosis

    5. Tissue Hypoxia

  • In what has become easy with chronic venous hyperemia:

    1. * Brown

    2. Cyanotic

    3. Red

    4. Gray

    5. Pale

  • In what part of clot from his organization

    1. * Head

    2. Body

    3. Tail

    4. It does not matter

    5. Back

  • In what vein thrombosis occurs local hepatic venous hyperemia:

    1. * Hepatic veins

    2. Hepatic artery

    3. Portal vein

    4. Cava-cava anastomoses

    5. Porto-cava anastomoses

  • Indicate how the disease occurs when bacterial embolism

    1. Atherosclerosis

    2. Malignant tumors

    3. * Sepsis

    4. Bends

    5. Fractures

  • Indicate the reason for the appearance of blood clots in the aortic aneurysm

    1. * Violation of the integrity of intima

    2. Thromboembolism

    3. Venous stagnation

    4. Expansion vessel

    5. Necrosis

  • Indicate where frequent hyaline thrombus

    1. In the aorta

    2. In the coronary arteries

    3. In the hollows aortic aneurysm

    4. In the hepatic artery

    5. * In vessels microcirculatory channel

  • Indicate which distinguish edema in the tissues, depending on the cause:

    1. * Kidney

    2. Hyaline

    3. Atrophic

    4. Glikogennye

    5. Hypertrophic

  • Inflammatory hyperemia occurs when:

    1. * Increase the number of histamine in tissues

    2. Increasing the number of eritrocits

    3. High blood pressure

    4. Decreased blood pressure

    5. Increasing venous pressure

  • It is called a consequence of thrombosis, in which cracks appear in the thrombus, upper room furnished epithelium

    1. * Sewage

    2. Petrification

    3. Epithelization
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