Modul 1General Pathomorphology Text tests



Download 0.93 Mb.
Page1/10
Date conversion14.05.2018
Size0.93 Mb.
  1   2   3   4   5   6   7   8   9   10
MODUL 1General Pathomorphology

Text tests

  1. All of the following diseases cause nephritic syndrome, EXCEPT:

    1. Lipoid nephrosis

    2. Diabetes mellitus

    3. Amyloidosis

    4. * Obesity

    5. Malignant disease.

  2. All of the following features characterize the bal looning degeneration of hepatocytes, EXCEPT:

    1. Swollen cells

    2. Edematous appearance

    3. Clumped cytoplasm

    4. * Large droplets of fat

    5. Large clear spaces.

  3. Ballooning degeneration of hepatocytes is caused by:

    1. * Accumulation of water and cellular swelling

    2. Retaining of biliary material

    3. Accumulation of iron or copp

    4. Accumulation of fat droplets

    5. Accumulation of lipofuscin.

  4. Ballooning degeneration of hepatocytes results from:

    1. * Viral hepatitis

    2. Alcoholic liver disease

    3. Biliary material accumulation

    4. Obesity

    5. Diabetes mellitus.

  5. Choose stage in necrosis morphogenesis:

    1. Paranecrosis irreversible change

    2. Coagulation

    3. Distrophy

    4. Autophagy

    5. * Cell death

  6. Choose stage in necrosis morphogenesis:

    1. Prinecrosis reversible change

    2. * Necrobiosis irreversible degenerative changes;

    3. Coagulation

    4. Distrophy

    5. Autophagy

  7. Choose stage in necrosis morphogenesis:

    1. * Paranecrosis reversible change

    2. Prinecrosis reversible change

    3. Coagulation

    4. Distrophy

    5. Autophagy

  8. Choose stage in necrosis morphogenesis:

    1. Paranecrosis irreversible change

    2. Coagulation

    3. Distrophy

    4. Autophagy

    5. * Cell autolysis

  9. Choose tipe of necrosis according to the cause:

    1. * Traumatic necrosis

    2. Coagulative necrosis

    3. Liquefactive necrosis

    4. Caseous necrosis

    5. Gangrenous necrosis

  10. Choose tipe of necrosis according to the cause:

    1. * Toxic necrosis

    2. Coagulative necrosis

    3. Liquefactive necrosis

    4. Caseous necrosis

    5. Gangrenous necrosis

  11. Choose tipe of necrosis according to the cause:

    1. * Trophoneurotic

    2. Coagulative necrosis

    3. Liquefactive necrosis

    4. Caseous necrosis

    5. Gangrenous necrosis

  12. Choose tipe of necrosis according to the cause:

    1. * Vascular

    2. Coagulative necrosis

    3. Liquefactive necrosis

    4. Caseous necrosis

    5. Gangrenous necrosis

  13. Choose tipe of necrosis according to the clinico-morphological forms:

    1. Vascular necrosis

    2. Toxic necrosis

    3. Trophoneurotic necrosis

    4. Traumatic necrosis

    5. * Coagulation (dry) necrosis

  14. Choose tipe of necrosis according to the clinico-morphological forms:

    1. Vascular necrosis

    2. Toxic necrosis

    3. Trophoneurotic necrosis

    4. Traumatic necrosis

    5. * Colliquative (liquefactive) necrosis

  15. Choose tipe of necrosis according to the clinico-morphological forms:

    1. Vascular necrosis

    2. Toxic necrosis

    3. Trophoneurotic necrosis

    4. Traumatic necrosis

    5. * Gangrene necrosis

  16. Coagulative necrosis is characterized by all of the following pathologic features, EXCEPT:

    1. Denaturation of cytoplasmic proteins

    2. Karyorrhaxis

    3. Karyopiknosis

    4. Breakdown of cell organelles

    5. * Lipid deposition

  17. Digestion of the cell by lysosome enzymes of immigrant leukocytes is termed:

    1. Autolysis

    2. Apoptosis

    3. * Heterolysis

    4. Inflammation

    5. Metaplasia.

  18. Early potentially reversible changes in myocardial cells induced by anoxia include all of the following conditions, EXCEPT:

    1. Failure of oxidative phosphorylation

    2. Depletion of ATP

    3. * Inhibition of anaerobic glycolysis and glycogenolysis

    4. Decrease in cellular pH

    5. Increase in intracellular Na and water and loss of intracellular K.

  19. Fatty change is seen by light microscopy as:

    1. Intracellular granules

    2. Basophilic granules

    3. Extracellular granules

    4. * Vacuoles in the cytoplasm around the nucleus

    5. Eosinophilic granules.

  20. Hepatic cell injury induced by CC14 is character ized by all of the following changes, EXCEPT:

    1. Lipid peroxidation

    2. Disaggregation of ribosomes

    3. * Depletion of intracellular lipids

    4. Influx of calcium

    5. Mitochondrial damage.

  21. Hie causes related to CCl4-induced hepatic cell injury are all of the following, EXCEPT.

    1. Processing by mixed function oxidases free radical formation

    2. Free radical formation lipid peroxidation of intracellular membranes

    3. * Failure of protein synthesis-disaggregation of ribosomes

    4. Decreased apolipoprotein synthesis intracel lular lipid accumulation

    5. Plasma membrane damage mitochondrial calcification.

  22. In long-standing hypertension and diabetes mellitus, the walls of arterioles, especially in the kidney, become:

    1. Serous

    2. Thined

    3. * Hyalinized

    4. Ulcered

    5. Pigmented.

  23. Necrotic cells are characterized by all of the following features, EXCEPT:

    1. Increased eosinophilia

    2. * Hyaline-like droplets in the cytoplasm

    3. Glassy appearance

    4. Lysis of cytoplasm

    5. Fragmentation of cytoplasm

  24. ?One of manifestations of metabolic derangements in cells is:

    1. Apoptosis

    2. * The intracellular accumulation of abnormal amounts of various substance and necrosis

    3. Hypertrophy and necrosis

    4. Metaplasia

    5. Atrophy

  25. One of the following changes in cells is apoptotic:

    1. Karyolysis

    2. * Nuclear pyknosis

    3. Plasmolysis

    4. Breakdown of mitochondrias

    5. Cellular swelling

  26. One of the following variants of necrosis can be found in myocardial infarct:

    1. * Coagulative necrosis

    2. Liquefactive necrosis

    3. Caseous necrosis

    4. Gangrenous necrosis

    5. Fat necrosis.

  27. One of the following variants of necrosis can be found in tuberculosis:

    1. * Caseous necrosis

    2. Gangrenous necrosis

    3. Liquefactive necrosis

    4. Fat necrosis

    5. Fibrinoid necrosis.

  28. One of the following variants of necrosis is associated with syphilis:

    1. Coagulative necrosis

    2. Liquefactive necrosis

    3. * Caseous necrosis

    4. Gangrenous necrosis

    5. Fat necrosis.

  29. One of the following variants of necrosis is associated with acute pancreatitis:

    1. Coagulative necrosis

    2. Liquefactive necrosis

    3. Caseous necrosis

    4. Gangrenous necrosis

    5. * Fat necrosis

  30. One of the possible causes of intracellular accumulation of metabolic substances and necrosis:

    1. * Genetic defects

    2. Inflammation

    3. Embolism

    4. Necrosis

    5. Activation of oncogenes.

  31. The following features characterize irreversible cell injury, EXCEPT:

    1. Nucleous pyknosis

    2. Karyolysis

    3. Karyorrhaxis

    4. * Endoplasmic reticulum swelling

    5. Mitochondrial swelling.

  32. The following features characterize reversible cell injury, EXCEPT:

    1. Blebs

    2. Endoplasmic reticulum swelling

    3. Dispersion of ribosomes

    4. * Myelin figures

    5. Autophagy

  33. The most common cause of fatty change in the heart is:

    1. Inflammation

    2. Neoplasia

    3. * Hypoxia

    4. Amyloidosis

    5. Autoimmune diseases.

  34. The nephrotic syndrome is characterized by all of the following manifestations, EXCEPT:

    1. Massive proteinuria

    2. Hypoalbuminemia

    3. Generalized edema

    4. Hyperlipidemia and lipiduria

    5. * Hyperalbuminemia.

  35. The stain used to identify fat is:

    1. Hematoxylin and eosin stain

    2. * Sudan III stain

    3. Congo red stain

    4. PAS reaction

    5. Metachromatic stain.

  36. The stain used to identify glycogen is:

    1. Hematoxylin and eosin stain

    2. Sudan III stain

    3. Congo red stain

    4. * PAS reaction

    5. Metachromatic stain.

  37. What stain is specific for iron:

    1. Hematoxylin and eosin

    2. Sudan III

    3. * Prussian blue reaction

    4. Congo red

    5. PAS-reaction.

  38. Which group of factors is most important in the cellular pathogenesis of irreversible cell injury?

    1. Lipid deposition, reduced protein synthesis, nuclear damage

    2. Mitochondrial condensation, glycolysis, sodium cell loss

    3. * Mitochondrial hyperplasia, lysozyme release, membrane injury

    4. Reduced ATP, increased calcium influx, membrane injury

    5. Ribosome detachment, glycolysis, nuclear damage.

  39. Which of the following events is most important in the development of irreversible cell injury?

    1. * Cell membrane damage

    2. Increased cell water

    3. Myelin figure accumulation

    4. Loss of ribosomes

    5. Swelling of mitochondria

  1. The pathological formation was revealed at the studying of the liver biopsy patterns. What is marked by the indicator on the illustration?

    1. The hypertrophied liver cells.

    2. Normal liver cells.

    3. * The granulation with the filament of the connecting tissue.

    4. The lipid including.

    5. The parenchyma of the liver.

  2. Pathologist explored the preparation of lymphatic node from a patient with a tuberculosis. What pathological process does a doctor see under the pointer?

    1. Exsudate edema of tissue

    2. Festering melting of tissue

    3. * Caseous necrosis of tissue

    4. Waxen candle (Zenker's necrosis)

    5. White pulp of lymphatic node

  3. The homogeneous masses in the center of microscope preparation of patient with tuberculosis testify about ...

    1. Serosal-hemorragic exudation in the lung tissue

    2. * Necrosis of lung tissue

    3. The inflammatory reaction in the lung tissue

    4. Metaplasy of the lung tissue

    5. Venous hyperemia and swollen

  4. The lung inflammation became the reason of death of 45-years-old man. A pathologist revealed the heat of the illness at the dissection. A lung is dense. What previous diagnosis will you put?

    1. Catarrhal inflammation.

    2. * Croupous inflammation.

    3. Hemorrhagic inflammation.

    4. Festering inflammation.

    5. Necrosis.

  5. At the section of thorax a pathologist found the rounded anomalous formation which are the soft consistency and which you see on illustration under a indicator. The diagnose is ...

    1. Tumour.

    2. * Abscess.

    3. Scar.

    4. Foreign body.

    5. Ordinary lung.

  6. The anomalous formation biopsy of the liver. What structure is marked by the indicator on the illustration.

    1. * The necrotic masses.

    2. The leucocytes infiltrates.

    3. The connecting tissue.

    4. The young granulation.

    5. The parenchyma of the liver.

  7. The material after appendectomy was investigated by the pathologist. What type of pathology do you see on the illustration?

    1. Exsudate inflammation.

    2. Proliferate inflammation.

    3. * Festering diffuse inflammation.

    4. Fibrinoid inflammation.

    5. Hemorrhagic exsudate inflammation.

  8. Pathologist investigated the byoptat (preparation of the tissue) from the inflammatory place of intestine. What structures does a pathologist see under a pointer?

    1. The connecting tissue of intestine with the inflammatory changes.

    2. Fatty tissue and leucocytes infiltrate.

    3. Festering melting.

    4. * Fibrinoid exsudate.

    5. Villi of intestine.

  9. The preparation of the tissue from the intestine was delivered for research. The pathologist sees under a pointer ...

    1. The connecting tissue of intestine with the inflammatory changes.

    2. Fatty tissue and leucocytes infiltrate.

    3. * Festering melting of tissue.

    4. Fibrinoid exsudate.

    5. Villi of intestine.

  10. The pathologist found the necrotizing areas of white color with connecting and granulation tissue around their (see illustration) at the investigation of the lung organ. Name the pathological process which is seen by the pathologist.

    1. Acute alterative inflammation

    2. Chronic heterospecific inflammatory process

    3. * Chronic specific inflammatory process

    4. Acute specific inflammatory process

    5. Necrosis of tissue

  11. Pathologist cut the lung of the dead man and discovered the small (size to 5mm) necrotizing areas of white color, that crumble easily (see the illustration). Name the most reliable diagnosis.

    1. Acute inflammation

    2. Necrotizing inflammation

    3. Chronic inflammation

    4. * Caseous necrosis (specific inflammation)

    5. Necrosis of tissue

  12. The dead man with kyphosis of lumbar portion of spine was discovered by the pathologyanatomist. The kyphosis developed during 3 years according to katamnesis (history of the disease). At the cutting pathologist saw the backbone which you see on the illustration. The pathologic areas are soft, white color and crumble easily. Your diagnosis...

    1. Osteodystrophy of the lumbar vertebrae.

    2. Traumatic damage of lumbar vertebrae.

    3. Benign tumor of vertebrae

    4. Malignant tumor of vertebrae

    5. * Caseous necrosis of vertebrae.

  13. The soft rounded pathological education, which you see on a photo, was found at a section in the left lung of 32-years-old man. Name the most reliable diagnosis.

    1. * A chronic abscess of lung.

    2. An acute abscess of lung.

    3. A scar.

    4. A hemorrhagic infarct.

    5. A neoplasm.

  14. The preparation of lymphatic node of tuberculosis patient was studied by pathologist. What pathological process does a doctor see on preparation? (See illustration)

    1. * Specific inflammatory process

    2. Unspecific (heterospecific) inflammatory process

    3. Acute disorders of blood circulation

    4. Forming of young granulation tissue

    5. Festering diffuse inflammation

  15. At research of lymphatic node from a patient with a tuberculosis a pathologist saw the structures which are marked by a pointer. Name these structures?

    1. Erythrocytes and hystiocytes

    2. Eosinophilic inflammatory infiltrate

    3. Pirogov-Langerhans giant cell

    4. * Hystio-leucocytes infiltrates

    5. Fibroblasts and granulocytes

  16. Pathologoanatomist found the structures in a lymphatic node of the tuberculosis patient (which are marked by a pointer on the illustration) at the microscope examination. Name this structures.

    1. Erythrocytes and hystiocytes

    2. Eosinophilic inflammatory infiltrate

    3. * Pirogov-Langerhans giant cell

    4. Hystio-leucocytes infiltrates

    5. Fibroblasts and granulocytes

  17. The abscess of lung was found at the pathoanatomical inspection. To define the type of inflammatory process with his original appearance.

    1. Proliferate inflammation.

    2. Serosal exsudate inflammation.

    3. Fibrinoid exsudate inflammation.

    4. Hemorrhagic exsudate inflammation.

    5. * Festering exsudate inflammation.

  18. The specific cells (which was found by the pathologist in the biopsy material (see an illustration)) gave the possibility to put the diagnose "rhinoscleroma". Whose scientist's name do these cells have?

    1. Bishout.

    2. Malory.

    3. Pirogov-Langerhans

    4. Virkhov.

    5. * Mykulich

  19. The intestine preparation was explored by the pathologoanatomist. What structures does he see under a pointer?

    1. * Glands of the intestine with the necrotizing and inflammatory changes.

    2. Hystio-leucocytes inflammatory infiltrate.

    3. Plural abscesses.

    4. Necrotizing areas.

    5. Villi of intestine.

  20. To define the type of inflammatory process with the real appearance of the trachea and bronchi surface (see the illustration).

    1. Festering inflammation.

    2. Hemorrhagic inflammation.

    3. Catarrhal inflammation.

    4. * Fibrinoid inflammation

    5. Mixed

  21. The pericardium structure changes was revealed at the dissection of man which died from cardiac illness. The consequences of which pathological process can you see?

    1. * Fibrinoid inflammation.

    2. Amiloidosis of the heart.

    3. Amiloidosis of the pericardium.

    4. Lipidosis of the heart.

    5. The neoplasm.

  22. The pathologist revealed the picture of inflammatory changes at the investigation of necropsy material (stain by haematoxillinum & eozinum). What is marked by the indicator on the illustration?

    1. Hystio-leucocytes infiltrates in the alveoli.

    2. * Hystio-leucocytes infiltrates in the parenchyma of lungs.

    3. A fibrin in the parenchyma of lungs.

    4. A fibrin in the alveoli.

    5. Necrosis of the pulmonary tissue.

  23. The pathology was revealed at the investigation of the liver biopsy. What is marked by the indicator on the illustration?

    1. The hypertrophied liver cells.

    2. The connecting tissue filaments.

    3. The inflammatory infiltrates around of abscess.

    4. Bloody lakes.

    5. * The saved normal parenchyma of the liver.

  24. Disease of lights became reason of the death of 30-years-old man. At research of necropsy material (stain by haematoxillinum & eozinum) a pathologist revealed the picture of inflammatory changes. What is marked by a indicator?

    1. Hystio-leucocytes infiltrates in the alveoli.

    2. Hystio-leucocytes infiltrates in the parenchyma of lungs.

    3. The fibrin filaments in the parenchyma of lungs.

    4. * The fibrin filaments in the alveoli.

    5. The erythrocytes in the vessels (hyperemia).

  25. Acute hepatitis is:

    1. fibrinous and necrotic

    2. alternatives and fibrinous

    3. and interstitial fibrinous

    4. None of the

    5. * etiology of exudative and productive

  26. After toxic dystrophy, viral or alcoholic hepatitis develops:

    1. postalkoholnyy cirrhosis

    2. total cirrhosis

    3. obstructive jaundice

    4. postvirusnyy cirrhosis

    5. * healed yellow

  27. All of the following diseases cause nephritic syndrome, EXCEPT:

    1. Lipoid nephrosis

    2. Diabetes mellitus

    3. Amyloidosis

    4. * Obesity

    5. Malignant disease.

  28. All of the following features characterize the bal looning degeneration of hepatocytes, EXCEPT:
  1   2   3   4   5   6   7   8   9   10


The database is protected by copyright ©dentisty.org 2016
send message

    Main page