1- Warm fluids, soft diet, analgesics, antipyretics and mouth wash.
2-Systemic antibiotics: penicillin for 7-10 days, if allergic to penicillin prescribes erythromycin.
2- Glandular fever (Infectious mononucleosis).
3- Scarlet fever.
1- Peritonsillar abscess.
2- Parapharyngeal and retropharyngeal abscesses.
3- Acute glomerulo-nephritis.
4- Acute rheumatic fever.
5- Acute otitis media.
1- Repeated attacks of acute tonsillitis (more than 5 attacks per year).
2- Obstructive sleep apnea.
3- Peritonsillar abscess (Quinsy).
4- Suspected tumour of the tonsil.
5- Recurrent attacks of rheumatic fever,
6- Attacks of acute glomerulonephritis,
7-As a part of other operations: Excision of long styloid process or glossopharyngeal neurectomy.
1- Bleeding tendency as haemophilia.
2- Acute upper respiratory tract infections.
3- Epidemics of poliomyelitis.
4- Cleft palate.
1- General anaesthesia: e.g. hypersensitivity to anaesthetic drugs or succinyl choline apnea.
2- Respiratory obstruction: This is the most serious complication; it may be due to early extubation, laryngeal spasm, inhalation of blood clots, vomitius or falling back of the tongue during recovery from anesthesia.
a) Primary: It is intra-operative blood loss. It is usually due to bad selection of the patient, bad dissection or injury of blood vessels.
b) Reactionary: Occurs within the first 24 hours after return from the theatre, it is usually due to oozing from the vessels after relaxation of the stretched blood vessels, slipping of a ligature, or rise of blood pressure during recovery from anesthesia.
c) Secondary: usually occurs between the 3rd and the 10th post operative days due to infection in the tonsillar fossa.
4- Pneumonia or lung abscess.
5- Trauma to the teeth, uvula, or the posterior pharyngeal wall.
6- Haematoma and edema of the uvula.
7- Infection of the tonsillar fossa, and acute otitis media.
8- Postoperative scarring of the palate.
9- Tonsillar remnant.
10- Subacute bacterial endocarditis.
Adenoid: (nasopharyngeal tonsil)
Mass of lymphoid tissue located in the upper and posterior wall of the nasopharynx, it is a normal structure with definite function namely the production of antibodies.
Physiological enlargement occurs at the age of 3 to 7 years. Pathological enlargement is due to inflammation during recurrent attacks of upper respiratory tract infection, which make it sufficient to produce symptoms, but the size of the adenoid in relative to size of the nasopharyngeal space is the more important factor in producing the symptoms.
Clinical picture: Enlargement of the adenoids will be sufficient to produce the following:
3- Post-nasal drip (mucoid or mucopurulent) which may leads to recurrent nocturnal cough.
4- Eustachian tube obstruction: predisposes to otitis media with effusion, recurrent attacks of acute otitis media.
5- Adenoid face: May develop with pinched nostrils, prominent incisors, open mouth, and high arched palate.
6-Mental dullness and apathy: due to day time sleepiness, hearing loss, chronic hypoxia.
1- Posterior rhinoscopy by post-nasal mirror examination: can show the adenoids but difficult especially in young children.
2- Endoscopic examination by fibro-optic nasopharyngeoscopy is the best method.
3- Lateral X-ray soft tissue for nasopharynx is good.
Enlargement of the adenoids sufficient to produce persist symptoms or recurs regularly.
Nasal obstruction which lead to mouth breathing, obstructive sleep apnea, snoring and sleep disturbance, adenoidal face, otitis media with effusion, recurrent acute otitis media.
Contraindications: similar to tonsillectomy.
Complications: Similar to tonsillectomy.
Other complications: include trauma to Eustachian cushions.